Say pathophysiology na ataki ed puso, a kabkabat met bilang myocardial infarction, et mangilalanor ed pakaderal na dala ya onagus ed parte na muscle na puso, a manresulta ed ipatey na saray selula na puso.
Saya so kaslakan a nagagawa lapud inkapot na coronary artery lapud blood clot, a mabetbet a resulta na atherosclerosis, sakey a kipapasen a say plaque et natitipon ed saray artery.
Say plaque et tugyopen na cholesterol, taba, cellular waste products, calcium, tan fibrin.
No omputok so plaque, nayarin nabuo so blood clot, ya ontakbong ed artery tan agmakasabi ed muscle na puso so dala ya napnoy oxygen.
Lapud kulang so oxygen, ompatey iray selula na muscle na puso, kanian onataki so puso.
Say laknab na impakasakitan so akadepende ed kabaleg na lugar a naaaruman na akasara ya artery tan say panaon manlapud inyataki anggad panagtambal.
Saray sintomas na ataki ed puso et nayarin sakit ed pagew, pakakaingas, panermen, pakaelew, tan sakit ed taklay, beklew, panga, odino beneg.
Say panagtambal ed ataki ed puso so kaslakan a mangilalanor ed pangipawil a tampol ed dala ed muscle na puso, balanglan diad panamegley na panagtambal odino saray proseso a singa say angioplasty tan stenting odino coronary artery bypass surgery.
Importante ya imanoen a say pathophysiology na ataki ed puso et komplikado tan nalalanor so dakel a sengegan, a singa say genes, estiloy kabibilay, tan kaliberliber.
Saray rason no akin ya onataki so puso et lapud atagey so blood pressure, atagey so cholesterol, panagsigarilyo, diabetes, sobran taba, kulang ya exercise, tan walay sakit ed puso na pamilya.
No nakontrol irayan sengegan, nabawasan so posibilidad a naataki ed puso.
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What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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