Pathophysiology ea kankere ea matšoafo e bolela liphetoho tse etsahalang lits'ebetsong le mekhoeng e tloaelehileng ea'mele e hlahang ho nts'etsopele le ho mpefala ha kankere ea matšoafo.
Kankere ea matšoafo ke lefu le rarahaneng le bakoang ke ho hōla le ho arohana ho sa laoleheng ha lisele tse sa tloaelehang matšoafong.
Lisele tsena li ka etsa lihlahala' me tsa namela likarolong tse ling tsa' mele, tsa baka matšoao le mathata a sa tšoaneng.
Pathophysiology ea kankere ea matšoafo e akarelletsa lintlha tse'maloa, ho akarelletsa liphetoho tsa liphatsa tsa lefutso, maemo a tikoloho le mekhoa ea bophelo.
Liphetoho tsa liphatsa tsa lefutso li ka etsahala DNA ea lisele tsa matšoafo, e leng se lebisang kholong e sa laoleheng ea lisele le ho arohana ha tsona.
Liphetoho tsena li ka futsetsoa kapa tsa fumanoa,'me li ka bakoa ke ho pepesehela lintho tse bakang kankere, tse kang mosi oa koae, radon, asbestos le tšilafalo ea moea.
Kankere ea matšoafo e ka aroloa ka mefuta e'meli e meholo: kankere ea matšoafo ea lisele tse nyane (SCLC) le kankere ea matšoafo e seng ea lisele tse nyane (NSCLC). NSCLC e boetse e arotsoe ka mefuta e meraro: adenocarcinoma, squamous cell carcinoma, le large cell carcinoma.
Pathophysiology ea mefuta ena ea kankere ea matšoafo e ka fapana, kaha e na le liphetoho tse fapaneng tsa liphatsa tsa lefutso'me e arabela ka tsela e fapaneng kalafong.
Pathophysiology ea kankere ea matšoafo e boetse e akarelletsa ho sebelisana pakeng tsa lisele tsa kankere le lisele tse e potolohileng, ho akarelletsa le tsamaiso ea'mele ea ho itšireletsa mafung.
Lisele tsa kankere li ka qoba tsamaiso ea'mele ea ho itšireletsa mafung, e leng se li lumellang hore li hōle le ho ata li sa thibeloe.
Ho phaella moo, tikoloho e nyenyane ea hlahala e ka khothalletsa ho hōla ha hlahala le metastasis ka ho fana ka tikoloho e tšehetsang lisele tsa kankere.
Pathophysiology ea kankere ea matšoafo ke mokhoa o rarahaneng le o matla,'me bafuputsi ba lula ba sebetsa ho utloisisa hamolemo mekhoa e ka sehloohong ea ho hlahisa mekhoa e sebetsang haholoanyane ea phekolo le ho ntlafatsa liphello tsa mokuli.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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['(5) polelo e saennweng ke wena, tlasa kotlo ya ho hlapanya leshano, ya hore tlhahisoleseding e tsebisong e nepahetse le hore o na le matla a ho tiisa ditokelo tsa mongodi tseo ho thweng di a tlolwa; ']
["le (6) ho saena ka letsoho kapa ka elektronike ha mong'a litokelo tsa bangoli kapa motho ea lumeletsoeng ho nka khato lebitsong la mong'a litokelo tsa bangoli. "]
['Ho hloleha ho kenyelletsa tlhahisoleseding yohle e ka hodimo ho ka baka ho dieha ha ho sebetswa tletlebo ya hao.']
['Ho Iteanya']
['Ka kōpo re romelle imeile ka potso / tlhahiso leha e le efe.']
What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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