Sa fisiopatologia de su diabete si riferit a s'istùdiu de sos protzessos fisiològicos disordinados chi batint a s'isvilupu de su diabete mellitu.
Su diabete mellitu est unu grupu de maladias metabolicas caraterizadas dae livellos artos de tzùcaru in su sàmbene (glucòsiu) chi risultant dae difetos in sa secretzione de s'insulina, in s'atzione de s'insulina, o in ambos.
Sa fisiopatologia de su diabete interessat s'interatzione cumplessa de fatores genèticos, ambientales e de istile de vida chi contribuint a s'isvilupu de sa maladia.
In su diabete de tipu 1, sa fisiopatologia interessat una distrutzione autoimmune de sas tzèllulas beta produtoras de insulina in su pancreas, chi giughet a una mèngua in sa produtzione de insulina.
Custu batit a un' incapatzidade de regulare sos livellos de glucosiu in su sàmbene, batende a un' iperglucèmia (zucru artu in su sàmbene).
In su diabete de tipu 2, sa fisiopatologia est prus cumplessa e interessat siat sa resistèntzia a s'insulina siat sa secretzione de s'insulina reduida.
Sa resistèntzia a s'insulina si verìficat cando sas tzèllulas de su corpus non risponent curretamente a s'insulina, batende a un'incapatzidade de impreare in manera eficatze su glucosiu pro s'energia.
Custu càusat chi su pancreas produat prus insulina pro proare a bìnchere sa resistèntzia, ma cun su tempus, su pancreas diat pòdere no èssere capatzu de sighire sa dimanda, giughende a una mèngua in sa produtzione de insulina.
Su diabete gestatzionale, chi si manifestat durante sa gravidade, est causadu dae càmbios ormones chi batint a sa resistèntzia a s'insulina.
Sa placenta produet ormones chi podent blocare s'atzione de s'insulina, giughende a un'aumentu de sos livellos de glucòsiu in su sàmbene.
Sa fisiopatologia de su diabete ìmplicat fintzas s'isvilupu de cumplicatziones, comente a sos dannos a sos vasos sanguignos, nervos e òrganos, chi podent batire a problemas graves de salude comente a maladias de su coro, atacu cardìaccu, maladias renales e pèrdida de sa bista.
Cumprèndere sa fisiopatologia de su diabete est fundamentale pro isvilupare tratamentos e istrategias de preventzione eficatzes pro custa maladia crònica.
Biochemistry and pathophysiology of diabetes. Proceedings of conference on pathophysiology and treatment of diabetes mellitus. 1990. Mol Cell Biochem. 1992, 109 (2): 97-204.
Surampudi PN, John-Kalarickal J, Fonseca VA: Emerging concepts in the pathophysiology of type 2 diabetes mellitus. Mt Sinai J Med. 2009, 76 (3): 216-26.
Johnson D: Selected pathophysiology of diabetes. Semin Perioper Nurs. 1998, 7 (3): 164-78.
Hirsch IB: The changing faces of diabetes. Prim Care. 2003, 30 (3): 499-510.
Guthrie RA, Guthrie DW: Pathophysiology of diabetes mellitus. Crit Care Nurs Q. , 27 (2): 113-25.
Felig P: Pathophysiology of diabetes mellitus. Med Clin North Am. 1971, 55 (4): 821-34.
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What is pathophysiology of diabetes?
Pathophysiology of diabetes refers to the study of the disordered physiological processes that lead to the development of diabetes mellitus.
Diabetes mellitus is a group of metabolic diseases characterized by high blood sugar (glucose) levels that result from defects in insulin secretion, insulin action, or both.
The pathophysiology of diabetes involves the complex interplay of genetic, environmental, and lifestyle factors that contribute to the development of the disease.
In type 1 diabetes, the pathophysiology involves an autoimmune destruction of the insulin-producing beta cells in the pancreas, leading to a deficiency in insulin production.
This results in an inability to regulate blood glucose levels, leading to hyperglycemia (high blood sugar).
In type 2 diabetes, the pathophysiology is more complex and involves both insulin resistance and impaired insulin secretion.
Insulin resistance occurs when the body's cells do not respond properly to insulin, leading to an inability to effectively use glucose for energy.
This causes the pancreas to produce more insulin to try to overcome the resistance, but over time, the pancreas may not be able to keep up with the demand, leading to a decrease in insulin production.
Gestational diabetes, which occurs during pregnancy, is caused by hormonal changes that lead to insulin resistance.
The placenta produces hormones that can block the action of insulin, leading to an increase in blood glucose levels.
The pathophysiology of diabetes also involves the development of complications, such as damage to blood vessels, nerves, and organs, which can lead to serious health problems like heart disease, stroke, kidney disease, and vision loss.
Understanding the pathophysiology of diabetes is crucial for developing effective treatments and prevention strategies for this chronic disease.
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