What is pathophysiology of Lung cancer?

['Iscurtade custa pàgina']

Cale est sa fisiopatologia de su tumore a sos purmones?

Sa fisiopatologia de su tumore a su purmones si riferit a sos cambiamentos in sos protzessos e mecanismos fisiològicos normales chi acontessent in s'isvilupu e sa progressione de su tumore a su purmones.

Su tumore a sos prumones est una maladia cumplessa chi naschet dae sa crèschida e divisione non controllada de tzèllulas anormales in sos prumones.

Custas tzèllulas podent formare tumores e si difundere a àteras partes de su corpus, giughende a vàrios sìntomos e cumplicaziones.

Sa fisiopatologia de su tumore a sos purmones interessat paritzos fatores, intre sos cales sas mutatziones genèticas, sos fatores ambientales e sas isceberas de istile de vida.

Sas mutatziones genèticas podent acontèssere in su DNA de sas tzèllulas de su purmones, giughende a una crèschida e divisione tzellulare non controllada.

Custas mutatziones podent èssere ereditàrias o achiridas, e podent èssere causadas dae s'espositzione a agentes cancerògenos, comente su fumu de su tabacu, su radon, s'asbestu e s'incuinamentu de s'àera.

Su tumore a su purmones si podet classificare in duos tipos printzipales: tumore a sas tzèllulas minores de su purmones (SCLC) e tumore a sas tzèllulas mannas de su purmones (NSCLC).

Sa fisiopatologia de custos tipos de tumore a sos prumones podet èssere diferente, ca tenent mutatziones genèticas diferentes e risponent a manera diferente a su tratamentu.

Sa fisiopatologia de su tumore a su purmones interessat fintzas s'interatzione intre sas tzèllulas tumorales e su tessidu a inghìriu, incluidu su sistema immunitàriu.

Sas tzèllulas tumorales podent fuire a su sistema immunitàriu, permitende·li de crèschere e si difundere sena controllu.

In prus, su microambiente de su tumore podet promòvere sa crèschida e sa metastasi de su tumore frunende un'ambiente favorèvole a sas tzèllulas tumorales.

Sa fisiopatologia de su tumore a sos purmones est unu protzessu cumplessu e dinàmicu, e sos chircadores sunt traballende in manera contina pro cumprèndere mègius sos mecanismos sutastantes pro isvilupare tratamentos prus eficatzes e megiorare sos resurtados de sos patzientes.

['Referèntzias']

PubMed/Medline https://www.nlm.nih.gov/databases/download/pubmed_medline.html

RefinedWeb https://arxiv.org/abs/2306.01116

Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.

Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.

Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.

Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .

Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .

Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.

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['Sos annùntzios depent èssere imbiados pro iscrittu pro mèdiu de posta eletrònica (càstia sa setzione "Cuntatu" pro s\'indiritzu de posta eletrònica). ']

["Sa DMCA recheret chi sa notìfica tua de sa presunta violatzione de copyright incluat sas informatziones sighentes: (1) descritzione de s'òpera amparada dae copyright chi est ogetu de sa presunta violatzione; (2) descritzione de su chi si narat chi est unu cuntènnidu chi diat pòdere èssere istadu violadu e informatziones bastantes pro nos permìtere de localizare su cuntènnidu; (3) informatziones pro ti cuntatare, intre cales s'indiritzu tuo, su nùmeru de telèfono e s'indiritzu de posta eletrònica; (4) una decraratzione tua pro nàrrere chi tenes bona fide chi su cuntènnidu de sa manera chi ses lamentende non est autorizadu dae su mere de su copyright, dae s'agente suo o dae cale si siat lege."]

["5) una decraratzione firma tua, suta pena de perjuria, chi is informatziones in sa notìfica sunt acuradas e chi tenes s'autorizatzione pro aplicare is deretos de autore chi sunt istados violados; "]

['e (6) una firma fìsica o eletrònica de su mere de su deretu de autore o de una persone autorizada a agire a nùmene suo. ']

['Si non ponis totu is informatzionis de prima podit essi chi su protzedimentu ti siat tardau.']

['Contatu']

['Manda·nos una mail cun cale si siat dimanda o sugerimentu.']

What is pathophysiology of lung cancer?

The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.

Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.

These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.

The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.

Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.

These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.

Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.

The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.

The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.

Cancer cells can evade the immune system, allowing them to grow and spread unchecked.

Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.

The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.

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Please note the neural net that generates answers to the questions, is specially inaccurate when it comes to numeric content. For example, the number of people diagnosed with a specific disease.

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