Patofisiologi kanker paru mengacu pada perubahan proses fisiologis normal wan mekanisme nang terjadi dalam perkembangan wan perkembangan kanker paru.
kanker paru adalah penyakit nang kompleks nang timbul dari pertumbuhan nang kada terkontrol lawan pambagian sel nang kada normal di paru.
Sel-sel ngini kawa mambantuk tumor wan manyabar ka bagian awak nang lain, manyabapakan bamacam gejala wan komplikasi.
Patofisiologi kanker paru malibatakan babarapa faktor, tamasuk mutasi genetik, faktor lingkungan, wan pilihan gaya hidup.
Mutasi genetik kawa tajadi di DNA sel paru-paru, nang manyabapakan partumbuhan wan pambagian sel nang kada taatur.
Mutasi ngini kawa diwarisakan atawa didapat, wan kawa disababakan oleh paparan karsinogen, kaya asap tembakau, radon, asbes, wan polusi udara.
kanker paru kawa diklasifikasikan jadi dua jenis utama: kanker paru sel halus (SCLC) wan kanker paru sel kada halus (NSCLC). NSCLC dibagi lagi jadi tiga subtipe: adenokarsinoma, karsinoma sel skuamosa, wan karsinoma sel ganal.
Patofisiologi dari jenis kanker paru-paru ini kawa berbeda, karena buhannya baisi mutasi genetik nang berbeda wan merespons secara berbeda lawan perawatan.
Patofisiologi kanker paru-paru jua malibatakan interaksi antara sel kanker wan jaringan sakitarnya, tamasuk sistem kekebalan.
sel kanker kawa menghindari sistem kekebalan tubuh, memungkinakan buhannya tumbuh lawan menyebar kada terkendali.
Salain itu, lingkungan mikro tumor kawa maningkatakan partumbuhan tumor wan metastasis lawan manyadiaakan lingkungan nang mandukung gasan sel kanker.
Patofisiologi kanker paru adalah proses nang kompleks wan dinamis, wan peneliti tarus bausaha gasan labih mamahami mekanisme nang mendasarinya gasan mangambangakan perawatan nang labih efektif wan maningkatakan hasil pasien.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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