Ti pathophysiology ti kanser ti prostate tuktukoyenna dagiti kangrunaan a mekanismo ken proseso a mangiturong iti itataud ken panagsaknap ti sakit.
Ti kanser ti prostate ket maysa a malignant tumor a tumaud kadagiti selula ti prostate gland, a bassit, sukog-walnut nga organo a masarakan iti baba ti basisaw kadagiti lallaki.
Ti prostate gland ket mangpataud iti seminal fluid, a mangtaraon ken mangyakar iti semilia.
Ti eksakto a pakaigapuan ti kanser ti prostate ket saan a naan-anay a maawatan, ngem adda sumagmamano a makagapu a mangpakaro iti posibilidad a maaddaan iti dayta a sakit.
Dagitoy iramanna ti edad, pakasaritaan ti pamilia, puli, ken sumagmamano a genetic mutation.
Ti kanser ti prostate ket ad-adda a gagangay kadagiti lallakay ken babbaket, a kaaduan kadagiti kaso ket mapasamak kadagiti lallaki nga agtawen iti nasurok a 65.
Kanayonanna, dagiti lallaki nga adda kapamiliada a nakaptan iti kanser ti prostate ket dakdakkel ti posibilidadna nga agsakit, kasta met dagiti Africano nga Americano ken dagiti lallaki a nagtaud iti Caribbean.
Ti pathophysiology ti kanser ti prostate ramanenna ti di makontrol a panagdakkel ken pannakabingay dagiti selula iti uneg ti prostate gland.
Mabalin a mapasamak daytoy gapu kadagiti genetic mutation a mangiturong iti sobra a panagtignay ti dadduma a tumataud a banag wenno ti di panagtignay dagiti tumor suppressor genes.
Dagitoy a mutation ti mabalin a mangibunga iti di naurnos a panagdakkel dagiti selula, nga agbanag iti panagbukel ti tumor.
Bayat a dumakdakkel ti tumor, mabalin a rautenna dagiti asideg a tisyu ken organo, kas iti basisaw, rectum, ken asideg a lymph node.
Iti dadduma a kaso, dagiti selula ti kanser mabalin a sumina iti kangrunaan a tumor ket agwarasda iti dadduma a paset ti bagi babaen ti dara wenno lymphatic system, maysa a proseso a maawagan metastasis.
No nagsaknapen ti kanser, narigrigat nga agasan.
Ti kanser ti prostate mabalin met a maapektaran kadagiti banag a mainaig iti hormone, nangruna ti androgen hormone a testosterone.
Ti testosterone ti mangtignay iti panagdakkel dagiti selula ti kanser ti prostate, ket adu a panangagas iti kanser ti prostate ti mangkissay iti kaadu daytoy a hormone wenno manglapped iti epektona.
Kas sumario, ti pathophysiology ti kanser ti prostate ramanenna ti di makontrol a panagdakkel ken pannakabingay dagiti selula iti uneg ti prostate gland, a mabalin a maimpluensiaan kadagiti genetic, hormonal, ken kasasaad iti aglawlaw.
Ti panangtarus kadagiti kangrunaan a pakaigapuan ti sakit ket napateg iti panangpataud kadagiti epektibo a panangagas ken panangpasayaat iti resulta dagiti pasiente nga addaan iti kanser ti prostate.
Zobniw CM, Causebrook A, Fong MK: Clinical use of abiraterone in the treatment of metastatic castration-resistant prostate cancer. Res Rep Urol. 2014, 6 (): 97-105.
Lim HY, Agarwal AM, Agarwal N, Ward JH: Recurrent epistaxis as a presenting sign of androgen-sensitive metastatic prostate cancer. Singapore Med J. 2009, 50 (5): e178-80.
Kohli M, Qin R, Jimenez R, Dehm SM: Biomarker-based targeting of the androgen-androgen receptor axis in advanced prostate cancer. Adv Urol. 2012, 2012 (): 781459.
Nelson JB, Hedican SP, George DJ, Reddi AH, Piantadosi S, Eisenberger MA, Simons JW: Identification of endothelin-1 in the pathophysiology of metastatic adenocarcinoma of the prostate. Nat Med. 1995, 1 (9): 944-9.
Msaouel P, Nandikolla G, Pneumaticos SG, Koutsilieris M: Bone microenvironment-targeted manipulations for the treatment of osteoblastic metastasis in castration-resistant prostate cancer. Expert Opin Investig Drugs. 2013, 22 (11): 1385-400.
Kotani K, Sekine Y, Ishikawa S, Ikpot IZ, Suzuki K, Remaley AT: High-density lipoprotein and prostate cancer: an overview. J Epidemiol. 2013, 23 (5): 313-9.
Jadvar H: Molecular imaging of prostate cancer: a concise synopsis. Mol Imaging. , 8 (2): 56-64.
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What is pathophysiology of prostate cancer?
The pathophysiology of prostate cancer refers to the underlying mechanisms and processes that lead to the development and progression of the disease.
Prostate cancer is a malignant tumor that arises from the cells of the prostate gland, which is a small, walnut-shaped organ located below the bladder in men.
The prostate gland produces seminal fluid, which nourishes and transports sperm.
The exact cause of prostate cancer is not fully understood, but several factors are known to increase the risk of developing the disease.
These include age, family history, race, and certain genetic mutations.
Prostate cancer is more common in older men, with the majority of cases occurring in men over the age of 65.
Additionally, men with a family history of prostate cancer are at an increased risk, as are African American men and men of Caribbean descent.
The pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland.
This can occur due to genetic mutations that lead to the overexpression of certain growth factors or the inactivation of tumor suppressor genes.
These mutations can result in the unregulated growth of cells, leading to the formation of a tumor.
As the tumor grows, it can invade nearby tissues and organs, such as the bladder, rectum, and nearby lymph nodes.
In some cases, cancer cells can break away from the primary tumor and spread to other parts of the body through the bloodstream or lymphatic system, a process known as metastasis.
Once the cancer has spread, it can be more difficult to treat.
Prostate cancer can also be influenced by hormonal factors, particularly the androgen hormone testosterone.
Testosterone can stimulate the growth of prostate cancer cells, and many treatments for prostate cancer aim to reduce the levels of this hormone or block its effects.
In summary, the pathophysiology of prostate cancer involves the uncontrolled growth and division of cells within the prostate gland, which can be influenced by genetic, hormonal, and environmental factors.
Understanding the underlying mechanisms of the disease is crucial for developing effective treatments and improving outcomes for patients with prostate cancer.
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