Pathophysiology ya saratani ya mapafu inahusu mabadiliko katika michakato ya kawaida ya kisaikolojia na mifumo ambayo hutokea katika maendeleo na maendeleo ya saratani ya mapafu.
Kansa ya mapafu ni ugonjwa tata unaosababishwa na ukuzi na mgawanyiko usio wa kudhibitiwa wa chembe zisizo za kawaida katika mapafu.
Chembe hizo zaweza kutokeza uvimbe na kuenea kwenye sehemu nyingine za mwili, na kusababisha dalili na matatizo mbalimbali.
Pathophysiology ya kansa ya mapafu inahusisha mambo kadhaa, kutia ndani mabadiliko ya chembe za urithi, mambo ya mazingira, na uchaguzi wa mtindo wa maisha.
Mabadiliko ya chembe za urithi yanaweza kutokea katika DNA ya chembe za mapafu, na kusababisha ukuzi na mgawanyiko wa chembe bila kudhibitiwa.
Mabadiliko hayo yanaweza kurithiwa au kupatikana, na yanaweza kusababishwa na vitu vinavyosababisha kansa, kama vile moshi wa tumbaku, radoni, asbesti, na uchafuzi wa hewa.
Kansa ya mapafu inaweza kugawanywa katika aina mbili kuu: kansa ya mapafu ya seli ndogo (SCLC) na kansa ya mapafu isiyo ya seli ndogo (NSCLC). NSCLC imegawanywa zaidi katika aina tatu ndogo: adenocarcinoma, carcinoma ya seli squamous, na carcinoma ya seli kubwa.
Pathophysiology ya aina hizi za saratani ya mapafu inaweza kutofautiana, kwani zina mabadiliko tofauti ya maumbile na hujibu kwa njia tofauti kwa matibabu.
Pathophysiology ya saratani ya mapafu pia inahusisha mwingiliano kati ya seli za saratani na tishu zinazozunguka, ikiwa ni pamoja na mfumo wa kinga.
Chembe za kansa zaweza kuepuka mfumo wa kinga, zikiruhusu zikue na kuenea bila kudhibitiwa.
Kwa kuongezea, mazingira madogo ya uvimbe yanaweza kukuza ukuaji wa uvimbe na metastasis kwa kutoa mazingira ya kuunga mkono seli za saratani.
Pathophysiology ya saratani ya mapafu ni mchakato tata na wenye nguvu, na watafiti wanafanya kazi kwa kuendelea kuelewa vizuri utaratibu wa msingi ili kuendeleza matibabu yenye ufanisi zaidi na kuboresha matokeo ya mgonjwa.
Li Z, Qian Y, Li W, Liu L, Yu L, Liu X, Wu G, Wang Y, Luo W, Fang F, Liu Y, Song F, Cai Z, Chen W, Huang W: Human Lung Adenocarcinoma-Derived Organoid Models for Drug Screening. iScience. 2020, 23 (8): 101411.
Nurwidya F, Syahruddin E, Yunus F: Pain management in lung cancer. Adv Respir Med. 2016, 84 (6): 331-336.
Khan KA, Kennedy MP, Moore E, Crush L, Prendeville S, Maher MM, Burke L, Henry MT: Radiological characteristics, histological features and clinical outcomes of lung cancer patients with coexistent idiopathic pulmonary fibrosis. Lung. 2015, 193 (1): 71-7.
Paramanantham A, Asfiya R, Das S, McCully G, Srivastava A: Extracellular Vesicle (EVs) Associated Non-Coding RNAs in Lung Cancer and Therapeutics. Int J Mol Sci. 2022, 23 (21): .
Lee D, Kim Y, Chung C: Scientific Validation and Clinical Application of Lung Cancer Organoids. Cells. 2021, 10 (11): .
Mucchietto V, Crespi A, Fasoli F, Clementi F, Gotti C: Neuronal Acetylcholine Nicotinic Receptors as New Targets for Lung Cancer Treatment. Curr Pharm Des. 2016, 22 (14): 2160-9.
Kanusho la dhima: matibabu
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What is pathophysiology of lung cancer?
The pathophysiology of lung cancer refers to the changes in the normal physiological processes and mechanisms that occur in the development and progression of lung cancer.
Lung cancer is a complex disease that arises from the uncontrolled growth and division of abnormal cells in the lungs.
These cells can form tumors and spread to other parts of the body, leading to various symptoms and complications.
The pathophysiology of lung cancer involves several factors, including genetic mutations, environmental factors, and lifestyle choices.
Genetic mutations can occur in the DNA of lung cells, leading to uncontrolled cell growth and division.
These mutations can be inherited or acquired, and they can be caused by exposure to carcinogens, such as tobacco smoke, radon, asbestos, and air pollution.
Lung cancer can be classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC is further divided into three subtypes: adenocarcinoma, squamous cell carcinoma, and large cell carcinoma.
The pathophysiology of these types of lung cancer can differ, as they have different genetic mutations and respond differently to treatment.
The pathophysiology of lung cancer also involves the interaction between cancer cells and the surrounding tissue, including the immune system.
Cancer cells can evade the immune system, allowing them to grow and spread unchecked.
Additionally, the tumor microenvironment can promote tumor growth and metastasis by providing a supportive environment for cancer cells.
The pathophysiology of lung cancer is a complex and dynamic process, and researchers are continuously working to better understand the underlying mechanisms to develop more effective treatments and improve patient outcomes.
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