Àrùn ọkàn, tí wọ́n tún ń pè ní myocardial infarction, máa ń jẹ́ kí ẹ̀jẹ̀ tó ń ṣàn lọ sí apá kan lára iṣan ọkàn dáwọ́ dúró, èyí sì máa ń mú kí àwọn sẹ́ẹ̀lì ọkàn kú.
Èyí sábà máa ń ṣẹlẹ̀ nítorí pé ẹ̀jẹ̀ kan tó ti di ẹ̀jẹ̀ máa ń dí ẹ̀jẹ̀ kan, èyí tó sábà máa ń jẹ́ àbájáde àrùn atherosclerosis, ìyẹn àìsàn tó máa ń mú kí ẹ̀jẹ̀ pọ̀ sí i nínú ẹ̀jẹ̀.
Àwọn èròjà tó ń jẹ́ cholesterol, èròjà ọ̀rá, èròjà tí kòkòrò máa ń tú jáde, èròjà calcium, àti èròjà fibrin ló máa ń wà nínú ẹ̀jẹ̀ náà.
Nígbà tí ẹ̀jẹ̀ náà bá fọ́, ó lè mú kí ẹ̀jẹ̀ dí, èyí tó lè dí ẹ̀jẹ̀ náà, tí kò sì ní jẹ́ kí ẹ̀jẹ̀ tó ní èròjà oxygen dé ọkàn.
Àìsí afẹ́fẹ́ yìí máa ń mú kí àwọn sẹ́ẹ̀lì iṣan ọkàn kú, èyí sì máa ń yọrí sí àrùn ọkàn.
Ìwọ̀n ibi tí ẹ̀jẹ̀ náà ti bà jẹ́ sinmi lórí bí ẹ̀jẹ̀ náà ṣe pọ̀ tó àti àkókò tó kọjá láàárín ìgbà tí ẹ̀jẹ̀ náà bà jẹ́ àti ìgbà tí wọ́n ṣe ìtọ́jú náà.
Lára àwọn àmì tí àrùn ọkàn máa ń fà ni ìrora tàbí ìrora tó máa ń wà ní àyà, àìsàn tí kì í jẹ́ kéèyàn lè mí dáadáa, àìsàn tí kì í jẹ́ kéèyàn lè mí dáadáa, àìsàn tí kì í jẹ́ kéèyàn lè mí dáadáa, àti ìrora tó máa ń wà ní apá, ọrùn, èékánná, tàbí ẹ̀yìn.
Ìtọ́jú àrùn ọkàn sábà máa ń ní í ṣe pẹ̀lú bí wọ́n ṣe máa mú kí ẹ̀jẹ̀ padà lọ sínú iṣan ọkàn kíákíá, bóyá nípa lílo oògùn tàbí nípa ṣíṣe iṣẹ́ abẹ bíi angioplasty àti stenting tàbí ṣíṣe iṣẹ́ abẹ tí wọ́n ń pè ní coronary artery bypass.
Ó ṣe pàtàkì láti kíyè sí i pé bí àrùn ọkàn ṣe máa ń wáyé máa ń díjú gan-an, ó sì máa ń dá lórí ọ̀pọ̀ nǹkan, títí kan àbùdá, ọ̀nà ìgbésí ayé, àti àyíká.
Lára àwọn nǹkan tó lè fa àrùn ọkàn ni bí ẹ̀jẹ̀ ṣe máa ń pọ̀ tó, bí èròjà cholesterol ṣe máa ń pọ̀ tó, bí sìgá ṣe máa ń jó, bí àrùn àtọ̀gbẹ ṣe máa ń pọ̀ tó, bó ṣe máa ń sanra tó, bí kò ṣe máa ń ṣe eré ìmárale, àti bí àìsàn ọkàn ṣe máa ń wáyé nínú ìdílé.
Ṣíṣe àbójútó àwọn kókó ewu wọ̀nyí lè ràn ẹ́ lọ́wọ́ láti dín ewu àrùn ọkàn kù.
Scott J: Pathophysiology and biochemistry of cardiovascular disease. Curr Opin Genet Dev. 2004, 14 (3): 271-9.
Liu Chung Ming C, Sesperez K, Ben-Sefer E, Arpon D, McGrath K, McClements L, Gentile C: Considerations to Model Heart Disease in Women with Preeclampsia and Cardiovascular Disease. Cells. 2021, 10 (4): .
Hansen J, Victor RG: Direct measurement of sympathetic activity: new insights into disordered blood pressure regulation in chronic renal failure. Curr Opin Nephrol Hypertens. 1994, 3 (6): 636-43.
LaMacchia JC, Roth MB: Aquaporins-2 and -4 regulate glycogen metabolism and survival during hyposmotic-anoxic stress in Caenorhabditis elegans. Am J Physiol Cell Physiol. 2015, 309 (2): C92-6.
Tham YK, Bernardo BC, Ooi JY, Weeks KL, McMullen JR: Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets. Arch Toxicol. 2015, 89 (9): 1401-38.
Lonn E: The clinical relevance of pharmacological blood pressure lowering mechanisms. Can J Cardiol. 2004, 20 Suppl B (): 83B-88B.
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Ìfọ̀rọ̀wérọ̀
Jọwọ fi imeeli ranṣẹ si wa pẹlu eyikeyi ibeere / imọran.
What is pathophysiology of heart attack?
The pathophysiology of a heart attack, also known as myocardial infarction, involves the disruption of blood flow to a part of the heart muscle, leading to the death of heart cells.
This typically occurs due to the obstruction of a coronary artery by a blood clot, which is often the result of atherosclerosis, a condition where plaque builds up in the arteries.
The plaque is made up of cholesterol, fatty substances, cellular waste products, calcium, and fibrin.
When a plaque ruptures, it can cause a blood clot to form, which can block the artery and prevent oxygen-rich blood from reaching the heart muscle.
This lack of oxygen causes the heart muscle cells to die, leading to a heart attack.
The extent of the damage depends on the size of the area supplied by the blocked artery and the time between the attack and treatment.
Symptoms of a heart attack can include chest pain or discomfort, shortness of breath, nausea, lightheadedness, and pain in the arms, neck, jaw, or back.
Treatment for a heart attack usually involves restoring blood flow to the heart muscle as quickly as possible, either through medication or procedures such as angioplasty and stenting or coronary artery bypass surgery.
It is important to note that the pathophysiology of a heart attack is complex and involves multiple factors, including genetic, lifestyle, and environmental factors.
Risk factors for heart attack include high blood pressure, high cholesterol, smoking, diabetes, obesity, lack of physical activity, and a family history of heart disease.
Managing these risk factors can help reduce the likelihood of experiencing a heart attack.
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